Puas yog Vincamine Powder muaj kev nyab xeeb rau kev siv?

Taw qhia ntawm Vincamine

Vincamine hmoovyog ib qho alkaloid muab rho tawm los ntawm Phlox tsob nroj Vincami-norL, uas tam sim no yog semi-synthetic raws li cerebrovascular dilator. Qhov kev npaj no muaj tartrate thiab Teprosilate ntxiv rau hydrochloride. Vincamine (VIN) belongs rau cov ntoo tawv dawb alkaloids hauv monoterpenoid indole alkaloids, uas tuaj yeem raug cais tawm ntawm Vincamine thiab Chemicalbook txiv kab ntxwv. Vinamine muaj lub luag haujlwm ntawm kev txo cov ntshav qabzib, tiv thaiv daim siab, tiv thaiv qog thiab tiv thaiv kab mob plawv thiab cerebrovascular. Txawm hais tias VIN tam sim no siv los ua cov ntsiav tshuaj thiab cov tshuaj maj mam tso tawm qeeb, nws cov qauv tshuaj lom neeg thiab cov khoom siv roj-philic ua rau nws cov dej solubility tsawg thiab qhov ncauj tsis zoo, uas txwv tsis pub nws daim ntawv thov kho mob. Cyclodextrin yog dav siv los ua tus neeg nqa tshuaj, uas tuaj yeem txhim kho cov dej solubility thiab ruaj khov ntawm cov tshuaj.Vinamine yog ib hom tshuaj, siv rau mob stroke, ischemic hypertensive encephalopathy, cerebral arterial sclerosis, cerebral ischemia, cerebral embolism, tsim nyog rau kev tshem tawm cov tsos mob ntawm progeric degeneration ntawm lub hlwb, xws li vertigo, mob taub hau, nco tsis tau, inattention, aphasia, Meniere's syndrome, thiab lwm yam. Nws kuj siv tau rau retinal hemorrhage, paj hlwb tachycardia thiab lwm yam vegetative paj hlwb.

Vincamine siv rau dab tsi?

1.Vinvinamine tuaj yeem dhau los ntawm cov ntshav-hlwb teeb meem los tswj thiab kho cov oxidative catabolism ntawm cov piam thaj hauv lub hlwb cov ntaub so ntswg ntawm cov kab mob, thiab normalize zus tau tej cov lactic acid thiab tso tawm carbon dioxide, yog li expanding cerebral me hlab ntsha thiab txhim kho cerebral ncig; Cov ntshav ntws hauv cov ntaub so ntswg hauv lub hlwb thiab lub hlwb ib txwm nyob ntawm tus neeg mob lub hlwb cov ntaub so ntswg tsis cuam tshuam loj heev, thiab cov hlab ntsha hauv lub cev tsis cuam tshuam. Kuj tseem muaj cov nyhuv sedative me me.

2. Lub mechanism ntawm vinvinamine nyob rau hauv kev kho mob ntawm ischemic cerebrovascular kab mob muaj xws li: kev txhim kho tus nqi ntawm kev siv cov pa oxygen thiab qabzib nyob rau hauv lub hlwb cov ntaub so ntswg; Dilate cerebrovascular thiab txhim kho microcirculation;

3. Thaiv calcium ion qeeb channel thiab voltage-gated sodium ion channel;

4. Anti-oxidation;

5. Tiv thaiv paj hlwb

Indications muaj feem xyuam rau cerebrovascular sclerosis, hlwb insufficiency thiab vascular kab mob ntawm lub qhov muag, pob ntseg, qhov ntswg thiab caj pas. Nws yog qhov tshwj xeeb tshaj yog haum rau cov kev tshwm sim thaum ntxov ntawm lub hlwb tsis ua haujlwm, xws li vertigo, mob taub hau, nco tsis tau, qaug zog, inattention thiab lwm yam. Nws kuj tseem tuaj yeem siv rau lub hlwb degeneration hauv cov neeg laus thiab nws cov tsos mob xws li kev ntxhov siab, kev ua phem, kev ntxhov siab, teeb meem ntawm lub cev muaj zog, kev xav qeeb, kev chim siab hauv cov neeg laus thiab cov kab mob sphincter, nrog rau kev mob tshwm sim thiab cov sequelae ntawm craniocerebral raug mob, xws li coma tom qab raug mob, cuam tshuam tsis nco qab, hemiplegia (hemiplegia), kev hais lus tsis meej, nco qab cov tsos mob ntawm lub hlwb raug mob thiab arterial dementia. Tsis tas li ntawd, nws kuj muaj qee yam cuam tshuam rau retinal vascular sclerosis, cochlear disorders los ntawm cov hlab ntsha, xws li kiv taub hau thiab Meniere syndrome.

Cov ntaub ntawv kev nyab xeeb

Cov kev phiv tshuaj Vincamine

1. Nws tsis tuaj yeem siv rau cov neeg mob qog nqaij hlav intracranial, nce intracranial siab, cov poj niam cev xeeb tub, lossis cov neeg mob uas muaj lub paj hlwb.

2. Rau cov neeg mob uas muaj myocardial infarction sequelae thiab cov organic teeb meem ntawm arrhythmia, ntau npaum li cas ntawm cov khoom no yuav tsum tau maj mam nce thiab electrogram xyuas yuav tsum tau siv yog tias tsim nyog.

3. Feem ntau ua tau zoo, xeev siab, ntuav, mob plab, cem quav thiab lwm yam kab mob plab, qee zaus tsis xis nyob, insomnia, urticaria. Kev txhaj tshuaj tuaj yeem ua rau hws ntau dhau.

Tab: 10 mg/tab. Kev txhaj tshuaj: 10mg / 2ml.

Aethromia-30 tau noj nrog 60mg ntawm kev npaj vinvinamine ib zaug. Cov concentration ntawm cov ntshav yog 12.5ug / ml rau 8 teev, thiab T1 / 2 yog 1 teev 40 feeb. Cov tshuaj concentration yog 17.5ug / ml, T1 / 2 yog kwv yees li 7 teev, thiab bioavailability yog kwv yees li 3 npaug ntawm kev npaj ntau. Cov khoom no tau noj 30mg txhua lub sijhawm, ob zaug ib hnub. Cov mob hnyav tuaj yeem nce mus rau 90mg / hnub. Tom qab 3-6 lub lis piam, cov tshuaj noj yog 30mg / hnub.

Yuav tiv tauj peb li cas?

Peb yog ib lub tuam txhab nrog ntau xyoo ntawm kev muag khoom nyob rau hauv cov nroj tsuag extract kev lag luam. Tom qab xyoo ntawm kev ua haujlwm, peb muaj ntau yam khoom, xws li txiv hmab txiv ntoo thiab zaub hmoov, tshuaj pleev ib ce, ntsiav tshuaj khoom qab zib thiab lwm yam khoom. Peb yog cov kws tshaj lij cov khoom siv raw khoom. Tam sim no, peb lub tuam txhab muag khoom pheej yigVincamine hmoov, uas yog dav siv hauv cov khoom kho mob, tshuaj, tshuaj pleev ib ce thiab lwm yam. Cov uas yuav tsum tau ua cov khoom kho mob tuaj yeem yuav cov khoom siv raw no. Lub caij no, raws li peb cov khoom raug muag ncaj qha los ntawm cov chaw tsim khoom, qhov zoo yog lav thiab koj tuaj yeem so assured mus yuav lawv.

Yog tias koj xav yuav lossis txaus siab rau nws, koj tuaj yeem xa email rau peb qhov chaw ua haujlwm emailella.zhang@huilinbio-tech.com iblos ntawm lub vev xaib thoob ntiaj teb. Peb yuav teb koj sai li sai tau thaum koj pom cov lus. Tsis tas li ntawd, koj tuaj yeem mus saib qhov chaw offline. Peb lub tuam txhab nyob hauv Chav 706, Tsev B, Fengze Science Park, No. 170, West Street, Yanta District, Xi 'an City, Shaanxi Province. Zoo siab txais tos koj tuaj xyuas.